Table 1 The prevalence of individual clinical features of both hypoadrenocorticism and atypical hypoadrenocorticism compiling data from the most relevant case series [14, 18, 25, 28, 29, 37, 38, 42, 46]. In most cases of hypoadrenocorticism, mineralocorticoid deficiency was presumed because of associated electrolyte abnormalities but this was not always the case. Some studies did not differentiate primary hypoadrenocorticism and glucocorticoid deficiency alone and clinical features were considered collectively and included in the hypoadrenocorticism group [18, 25, 37, 38]. In most cases of “atypical” hypoadrenocorticism, electrolyte abnormalities were not present and while glucocorticoid deficiency was confirmed, mineralocorticoid production was not usually evaluated. Secondary hypoadrenocorticism was confirmed in only a small number of these cases

From: Diagnosis of canine spontaneous hypoadrenocorticism